Teen dies of stroke after love bite

Love bite stroke hickeyA teenager has died after a love bite from his girlfriend caused a blood clot that quickly led to a stroke.

Julio Macias Gonzalez, a 17-year-old from Mexico City, raised alarm among his family when he began convulsing at the dinner table.

It is thought his girlfriend gave him a hickey earlier that evening which caused a blood clot that travelled to the teen’s brain. Paramedics were called to the scene but Julio could not be saved and died shortly after.

The young man’s family are blaming his 24-year-old girlfriend for his death but she has now disappeared.

It is not the first time a love bite has been believed to have triggered a reaction. In 2011 a 44-year-old woman in New Zealand lost movement in her left arm after having a stroke.

On noticing a faded love bite, doctors quickly realised damage to a major artery in her neck and linked it to her paralysis. The suction had caused a blood clot to form which then travelled to the woman’s heart, causing a stroke.

Dr Teddy Wu, who treated the woman at Auckland’s Middlemore Hospital, said: “To my knowledge, it’s the first time someone has been hospitalised by a hickey.”

Love bites or hickeys are caused by a person sucking on an area of another person’s skin, more commonly the neck. The suction causes blood vessels under the skin to burst which causes bruising that can last up to two weeks.

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Why all migraine patients should be treated with magnesium

magnesiumMagnesium, the second most abundant intracellular cation, is essential in many intracellular processes and appears to play an important role in migraine pathogenesis.

Routine blood tests do not reflect true body magnesium stores since <2% is in the measurable, extracellular space, 67% is in the bone and 31% is located intracellularly. Lack of magnesium may promote cortical spreading depression, hyperaggregation of platelets, affect serotonin receptor function, and influence synthesis and release of a variety of neurotransmitters.

Migraine sufferers may develop magnesium deficiency due to genetic inability to absorb magnesium, inherited renal magnesium wasting, excretion of excessive amounts of magnesium due to stress, low nutritional intake, and several other reasons.

There is strong evidence that magnesium deficiency is much more prevalent in migraine sufferers than in healthy controls. Double-blind, placebo-controlled trials have produced mixed results, most likely because both magnesium deficient and non-deficient patients were included in these trials. This is akin to giving cyanocobalamine in a blinded fashion to a group of people with peripheral neuropathy without regard to their cyanocobalamine levels.

Both oral and intravenous magnesium are widely available, extremely safe, very inexpensive and for patients who are magnesium deficient can be highly effective. Considering these features of magnesium, the fact that magnesium deficiency may be present in up to half of migraine patients, and that routine blood tests are not indicative of magnesium status, empiric treatment with at least oral magnesium is warranted in all migraine sufferers.

Reference:

Mauskop, A., et al., Why all migraine patients should be treated with magnesium. J Neural Transm, 2012. 119 (5): p. 575-9.


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